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NLR

结构式 货号 产品名称 CAS号
Mifamurtide sodium hydrate 结构式
BCP49623 米伐木肽 838853-48-8
Mifamurtide sodium hydrate is the sodium hydrate of mifamurtide. Mifamurtide is a nonspecific immunomodulator that acts by stimulating immune responses by activating macrophages and monocytes. Mifamurtide is a specific ligand for NOD2 and is used as an insulin sensitizer and may also be used in osteosarcoma research.
QS-21 结构式
BCP49573 QS-21 141256-04-4
QS21 is a purified, natural saponin isolated from the soapbark tree Quillaja saponaria Molina with potential immunoadjuvant activity. When co-administered with vaccine peptides, QS21 may increase total antitumoral vaccine-specific antibody responses and cytotoxic T-cell responses.
Selnoflast 结构式
BCP49321 Selnoflast 2260969-36-4
Selnoflast is a NLRP3 inhibitor.
Emlenoflast 结构式
BCP35616 Emlenoflast 1995067-59-8
Emlenoflast is a potent and selective inhibitor of NLRP3 inflammasome.
MCC7840 sodium 结构式
BCP48756 MCC7840 sodium 2380032-29-9
MCC7840 sodium, a sulfonylurea, is a potent and selective inhibitor of NLRP3 inflammasome, with an IC50 of <100 nM. MCC7840 sodium can be used for the research of inflammatory diseases.
BMS-986299 结构式
BCP44085 BMS-986299 2242952-69-6
BMS-986299 is a First-In-Class NLRP3 Innate Agonist with Potent Antitumor Activity.
NOD-IN-1 结构式
BCP24808 NOD-IN-1 132819-92-2
NOD-IN-1 is a potent mixed inhibitor of nucleotide-binding oligomerization domain (NOD)-like receptors, NOD1 and NOD2, with IC50 of 5.74 μM and 6.45 μM, respectively.
Troxerutin 结构式
BCP13421 曲克芦丁 7085-55-4
Troxerutin, also known as vitamin P4, is a tri-hydroxyethylated derivative of natural bioflavonoid rutins which can inhibit the production of reactive oxygen species (ROS) and depress ER stress-mediated NOD activation.
Arglabin 结构式
BCP09930 阿格拉宾 84692-91-1
Arglabin is a sesquiterpene gamma-lactone is isolated from Artemisia glabella; anticancer natural compound.
Nodinitib-1 结构式
BCP20383 Nodinitib-1 799264-47-4
Afuresertib is an orally available, ATP-competitive, pan-AKT inhibitor with Ki of 0.08, 2 and 2.6 nM against AKT1, AKT2 and AKT3, respectively.
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