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TNF Receptor

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TNF Receptor

结构式 货号 产品名称 CAS号
ABD328 结构式
BCP49194 ABD328 1055896-19-9
ABD328 is an orally active, small molecule inhibitor of RANKL and TNF signalling that inhibits inflammation in vivo.
UTL-5g 结构式
BCP48944 UTL-5g 646530-37-2
UTL-5g (GBL-5g), an anti-inflammatory TNF-α inhibitor, has chemoprotective and liver radioprotective effects. UTL-5g lowers hepatotoxicity, nephrotoxicity, and myelotoxicity induced by Cisplatin through TNF-α inhibition among other factors.
2'-Fucosyllactose 结构式
BCP47768 2'-岩藻糖基乳糖 41263-94-9
2'-Fucosyllactose is an oligosaccharide that could be derived from human milk.
ONC-201 Dihydrochloride 结构式
BCP46830 ONC-201 Dihydrochloride 1638178-82-1
ONC-201, also known as TIC10, is a potent, orally active, and stable small molecule that transcriptionally induces TRAIL in a p53-independent manner and crosses the blood-brain barrier.
AN0128 结构式
BCP44562 AN0128 872044-70-7
AN0128 is a tumour necrosis factor alpha (TNF-α) inhibitor potentially for the treatment of atopic dermatitis.
UCB-9260 结构式
BCP40717 UCB-9260 1515888-53-5
UCB-9260 is an orally active inhibitor that inhibits TNF signalling by stabilising an asymmetric form of the trimer.
(Rac)-Benpyrine 结构式
BCP37768 (Rac)-Benpyrine 1333714-43-4
(Rac)-Benpyrine is a racemate of Benpyrine. (Rac)-Benpyrine is a potent and orally active TNF-α inhibitor and has the potential for TNF-α mediated inflammatory and autoimmune disease research.
Corilagin 结构式
BCP25487 柯里拉京 23094-69-1
Corilagin, discovered in many medicinal plants such as Phyllanthus speices etc., has multiple activities including anti-oxidative, anti-inflammatory, anti-apoptotic, hepatoprotective and others. It is an inhibitor of TNF-α.
Hispidol 结构式
BCP32790 Hispidol 5786-54-9
Hispidol is a potential therapeutic for inflammatory bowel disease; inhibits TNF-α induced adhesion of monocytes to colon epithelial cells with an IC50 of 0.50 µM.
Methylthiouracil 结构式
BCP21374 甲基硫脲嘧啶 56-04-2
Methylthiouracil is an antithyroid agent. Methylthiouracil suppresses the production TNF-α and IL-6, and the activation of NF-κB and ERK1/2.
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